Welsh Journals

Clarke, C. A., McConnell, R. B., Sheppard, P. M. and Woodrow, J. C. (1966). Prevention of Rh-haemolytic disease. Brit. Med. J., II, 1074. Clarke, C. A. and Sheppard, P. M. (1966). A local survey of the distribution of industrial melanic forms in the moth Bistonbetulariaand estimates of the selective values of these in an industrial environment. Proc. Roy. Soc. B., 165, 424-439. Das, K. M., Eastwood, M. A., McManus, J. P. A. and Sircus, W. (1973). Adverse reactions during salicylazosulfa- pyridine therapy and the relation with drug metabolism and acetylator phenotype. New Engl. J. Med., 289, 491-495. Devadatta, S., Gangadharam, R. H., Andrews, R. H., Fox, W., Ramakrishnan, C. V., Selkon, J. B. and Velu, S. (1960). Peripheral neuritis due to ironiazid. Bull. World Health Org., 23, 587-598. Doll, R. and Buch, J. (1950). Hereditary factors in peptic ulcer. Ann Eugen (London), 15, 135-146. Ehlers, N., Kissmeyer-Nielsen, F., Kjerbye, K. E. and Lamm, L. U. (1974). HL-A27 in acute and chronic uveitis. Lancet, I, 99. Edwards, J. H. (1965). The meaning of the associations between blood groups and disease. Ann. Hum. Genet., 29, 77-83. Emery, A. E. and Lawrence, J. S. (1967). Genetics of ankylosing spondylitis. J. Med. Genet., 4, 239-244. Evans, D. A. P. (1963). Agweddau etifeddegol cyffuriau. Y Gwyddonydd, 4, 165-174. Evans, D. A. P. (1973). Coeliac Disease and HL-A8. Lancet, II, 1096. Evans, D. A. P., Davison, K. and Pratt, R. T. C. (1965). The influence of acetylator phenotype on the effects of treating depression with phenelzinc. Clin. Pharmacol. Therap., 6, 430-435. Evans, D. A. P., Horwich, L., McConnell, R. B. and Bullen, M. F. (1968). The influence of the ABO blood groups and secretor status on bleeding and on perforation of a duodenal ulcer. Gut, 9, 319-322. Evans, D. A. P. and White, T. A. (1964). Human acetylation polymorphism. J. Lab. Clin. Med., 63, 394-403. Eze, L. C. and Evans, D. A. P. (1973). Canlyniadau nas cyhoeddwyd. Finn, R., Clarke, C. A., Donohoe, W. T. A., McConnell, R. B., Sheppard, P. M., Lehane, D. and Kulke, W. (1961). Experimental studies on the prevention of Rh-haemolytic disease. Brit. Med. J., I, 1486-1490. Ford, E. B. (1940). Polymorphism and taxonomy. In New Systematics, ed. J. Huxley, Clarendon Press, Oxford, p. 493, quoted in: Genetic Polymorphism, Faber and Faber, London, 1965. Friedman, C. (1969). Rh-Isoimmunization and Erythro- blastosis Fetalis, Butterworths, London. Geldmacher von MaIlinckrodt, M., Lindorf, H. H., Petenyi, M., Flugel, M., Fischer, T. and Hiller, T. (1973). Genetisch determinierter polymorphismus der menschlich n serum-paroxonase (EC 3.1,1 .2.), Humangenetik, 17, 33 1-3. s. Greaves, J. (1972). How super oats survive, New Scienti < 56, 156-158. Horwich, L., Evans, D. A. P., McConnell, R. B. ai-J Donohoe, W. T. A. (1966). ABO blood groups in gasti bleeding. Gut, 7, 680-685. Joysey, V. C., Roger, J. H., Evans, D. B. and Herbertso i, B. M. (1973). Kidney graft survival and matching for HL and ABO antigens. Nature, 246, 163-165. Kellerman, G., Luyten-Kellerman, M. and Shaw, C. R. (1973a). Genetic variation of Aryl Hydrocarbon Hydroxylase in Human Lymphocytes. Amer. J. Hum. Genet., 25, 327-33 i. Kellerman, G., Shaw, C. R. and Luyten-Kellerman, M. (1973b). Aryl hydrocarbon hydroxylase inducibility and bronchogenic carcinoma. New Engl. J. Med., 289, 934-937. Landsteiner, K. (1901). Uber Agglutinationserscheinungen normalen menslichen Blutes. Wein klin Wschr, 14 1132-1134. Landsteiner, K. and Weiner, A. S. (1940). An agglutinable factor in human blood recognized by immune sera for rhesus blood. Proc. Soc. Exp. Biol., N.Y., 43, 223. Levine, P. (1943). Serological factors as possible causes in spontaneous abortions. J. Hered., 34, 71-80. Levine, P., Katzin, E. M. and Burnham, L. (1941). Iso-immunization in pregnancy. Its possible bearing on the etiology of erythroblastosis fetalis. J.A.M.A., 116, 825-827. McConnell, R. B. and Sheppard, P. M. (1956). The secretor character and disease. Acta genet., 4, 574-579. Menon, N. K. (1968). Madras study of supervised once- weekly chemotherapy. Bull. Internat. Union against Taberc, 41, 316-321. Nevanlinna, H. R. and Vainio, T. (1956). The influence of mother-child ABO incompatibility on Rh-immunisation. Vox sang (Basel), 1, 26-36. O'Reilly, R. A. (1970). The second reported kindred with hereditary resistance to oral anticoagulant drugs. New Engl. J. Med., 282, 1448-1451. O'Reilly, R. A., Aggeler, P. M., Hoag, L. S., Leong, L. S. and Kropatkin, M. L. (1964). Hereditary transmission of exceptional resistance to coumarin anticoagulant drugs. The first reported kindred. New Engl. J. Med., 271, 809-815. Perry, H. M., Tan, E. M., Carmody, S. and Sakamoto, A. (1970). Relationship of acetyl transferase activity to antinuclear antibodies and toxic symptoms in hypertensive patients treated with hydralazinc. J. Lab. Clin. Med., 76, 114-125. Tokuhata, G. K. and Lilienfeld, A. M. (1963). Familial aggregation of lung cancer in humans. J. Nat. Cancer Inst., 30, 289-312. Woodrow, J. C. (1974). Rh-immunization and its prevention. Pathobiology Annuals, 4 (in the press). Woolf, B. (1955). On estimating the relation between blood group and disease. Ann. Hum. Genet., 19, 251-253.